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Environmental or self-antigens and homotypic interactions result in BCR and Toll-like receptor (TLR) signaling, amplifying the reaction of CLL cells to other signals from the microenvironment and escalating the activation of anti-apoptotic and proliferation pathways.
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mutations provided The truth that, as defined down below, CLL therapy relies within the existence or absence of these mutations. The current consensus is that, apart from clonal mutations, subclonal mutations which has a variant allelic frequency ranging from five to 10% (and therefore underneath the threshold of detection by common molecular approaches) could also be claimed, While Individuals that has a variant allelic frequency lower than 5% shouldn't, but there is Substantially controversy around these concerns which suggestion may well alter Later on.
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